Ventricular tachycardia: Difference between revisions

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*VT more likely than SVT - AV dissociation (lack of synchronicity between atria and ventricles); fusion beat (where normal and ectopic ventricular beats coincide); QRS > 40ms; [[ECG|rS pattern]] (small rise, big fall, back to normal instead of QRS complex) in V1.
*VT more likely than SVT - AV dissociation (lack of synchronicity between atria and ventricles); fusion beat (where normal and ectopic ventricular beats coincide); QRS > 40ms; [[ECG|rS pattern]] (small rise, big fall, back to normal instead of QRS complex) in V1.
*SVT more likely than VT - [[RBBB]] pattern; varying bundle branch block; qR pattern (no initial drop in QRS complex) in V1; ectopic P wave preceding arrhythmia.
*SVT more likely than VT - [[RBBB]] pattern; varying bundle branch block; qR pattern (no initial drop in QRS complex) in V1; ectopic P wave preceding arrhythmia.
====Bloods====
*[[U+Es]] - particularly calcium, potassium and magnesium.
*Levels of therapeutic drugs like digoxin
*Trop T for MI
*[[CXR]] - due to possibility of heart  failure


===Management===
===Management===
===Prognosis===
===Prognosis===

Revision as of 17:44, 7 December 2009

Definition

This is a broad-complex tachycardia (QRS >120ms/3 small squares) which originates in the ventricles.

Epidemiology

Common.jpgReally common and dangerous, particularly in coronary artery disease.

Pathophysiology

There are three different types of VT:

  • Fascicular - uncommon; associated with structural heart defects; from left bundle branch; short QRS complexes (0.11-0.14s); QRS complexes look like RBBB
  • Right ventricular outflow tract - right axis deviation; LBBB pattern; provoked by catecholamine release, sudden changes in heart rate, exercise; responds to alpha-blockers and calcium antagonists.
  • Polymorphic - looks like Torsades de pointes (QT interval normal in sinus rhythm); similar to AF; can lead to cardiogenic shock and VF.
  • Torsades de pointes

Risk Factors

Clinical Features

Presents with features of ischaemic heart disease or haemodynamic compromise. Chest pain, palpitations, dyspnoea, dizziness, syncope and possibly symptoms of heart failure.

Investigations

ECG

Differences between VT andd SVT:

  • VT more likely than SVT - AV dissociation (lack of synchronicity between atria and ventricles); fusion beat (where normal and ectopic ventricular beats coincide); QRS > 40ms; rS pattern (small rise, big fall, back to normal instead of QRS complex) in V1.
  • SVT more likely than VT - RBBB pattern; varying bundle branch block; qR pattern (no initial drop in QRS complex) in V1; ectopic P wave preceding arrhythmia.

Bloods

  • U+Es - particularly calcium, potassium and magnesium.
  • Levels of therapeutic drugs like digoxin
  • Trop T for MI
  • CXR - due to possibility of heart failure

Management

Prognosis